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Pat Schoff, Ph.D., Research Associate
Position and Focus
Current research is focusing on ecological toxicology with a specific focus on the prairie pothole region of the Midwest identifying amphibian indicators of ecosystem health.
Postdoctoral Institute for Enzyme Research, University of Wisconsin, Madison, 1982-86
Ph.D., Zoology and Physiology, University of Wyoming, 1982
B.S. Biology, University of Wisconsin, Stevens Point, 1976
7: Fitzpatrick, Patricia Fonseca, Alberto & McAllister, Mary Louise. 2011. From the Whitehorse Mining Initiative Towards Sustainable Mining: lessons learned. Journal of Cleaner Production 19:376--384.
7: Rohr, J R, Schotthoefer, A M, Raffel, T R, Carrick, H J, Halstead, N, Hoverman, J T, Johnson, C M, Johnson, L B, Lieske, C, Piwoni, M D Schoff, P K & Beasley, V R. 2008. Agrochemicals increase trematode infections in a declining amphibian species. Nature 455:1235--1239.
7: Brooks, R P, Patil, G P, Fei, S, Gitelman, A I Myers, W L & Reavie, E D. 2007. The next generation of ecological indicators of wetland condition. Ecohealth 4:176--178.
7: Schoff, P K & Ankley, G T. 2004. Effects of methoprene, its metabolites, and breakdown products on retinoid-activated pathways in transfected cell lines. Environmental Toxicology and Chemistry 23:1305--1310.
7: Schoff, P K, Johnson, C M, Schotthoefer, A M, Murphy, J E, Lieske, C, Cole, R A Johnson, L B & Beasley, V R. 2003. Prevalence of skeletal and eye malformations in frogs from north-central United States: estimations based on collections from randomly selected sites. Journal of Wildlife Diseases 39:510--521.
to view complete publication list.
- Pat Schoff CV
Project list for Pat Schoff :
(A link will go to the project's current report, an arrow will take you to a project's home page)
Gonadal Deformities in Smallmouth Bass as Indicators of Endocrine Disruption in the St. Louis River Estuary
This project focuses on the background occurrence of one potential biomarker of endocrine disruption in smallmouth bass, the presence of oocytes in testicular tissue, and evaluates the likelihood that any elevated prevalence of this deformity could be caused by exposure to synthetic estrogen during early life stages.